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mitochondrial antioxidants mitoq  (MitoQ Ltd)

 
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    MitoQ Ltd mitochondrial antioxidants mitoq
    Mitochondrial Antioxidants Mitoq, supplied by MitoQ Ltd, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/mitochondrial antioxidants mitoq/product/MitoQ Ltd
    Average 90 stars, based on 1 article reviews
    mitochondrial antioxidants mitoq - by Bioz Stars, 2026-02
    90/100 stars

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    Transcriptional reprogramming of ROS signaling in c Sirt4 -Tg mice following TAC. Changes in expression of genes encoding for proteins involved in A) ROS production as determined by RNA-sequencing (n = 5–6) and B) validation of Nox4 expression by RT-qPCR ( n = 9–10); and C) <t>antioxidant</t> stress defense as determined by RNA-sequencing (n = 5–6) and D) validation of Gpx1 expression by RT-qPCR ( n = 9–10) in hearts of Control and c Sirt4 -Tg mice 12 weeks following TAC surgery with or without MitoQ supplementation. 2-Way ANOVA: #, effect of genotype; §, effect of treatment; %, effect of interaction. * p < 0.05; **p < 0.01; ***p < 0.001; ****p < 0.0001 using Fisher's LSD test. CAT, catalase; c Sirt4 -Tg, cardiomyocyte-specific overexpression of Sirt4 ; CYBA, cytochrome b -245, alpha chain; CYBB, cytochrome b -245, beta chain; GPX, glutathione peroxidase; MAOA, monoamine oxidase A; MAOB, monoamine oxidase B; MitoQ, mitoquinone; NOX4, NADPH oxidase 4; PRDX3, peroxiredoxin 3; SOD2, superoxide dismutase 2, mitochondrial; TAC, transverse aortic constriction; TMX, thioredoxin-related transmembrane protein; XDH, xanthine dehydrogenase.
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    Transcriptional reprogramming of ROS signaling in c Sirt4 -Tg mice following TAC. Changes in expression of genes encoding for proteins involved in A) ROS production as determined by RNA-sequencing (n = 5–6) and B) validation of Nox4 expression by RT-qPCR ( n = 9–10); and C) <t>antioxidant</t> stress defense as determined by RNA-sequencing (n = 5–6) and D) validation of Gpx1 expression by RT-qPCR ( n = 9–10) in hearts of Control and c Sirt4 -Tg mice 12 weeks following TAC surgery with or without MitoQ supplementation. 2-Way ANOVA: #, effect of genotype; §, effect of treatment; %, effect of interaction. * p < 0.05; **p < 0.01; ***p < 0.001; ****p < 0.0001 using Fisher's LSD test. CAT, catalase; c Sirt4 -Tg, cardiomyocyte-specific overexpression of Sirt4 ; CYBA, cytochrome b -245, alpha chain; CYBB, cytochrome b -245, beta chain; GPX, glutathione peroxidase; MAOA, monoamine oxidase A; MAOB, monoamine oxidase B; MitoQ, mitoquinone; NOX4, NADPH oxidase 4; PRDX3, peroxiredoxin 3; SOD2, superoxide dismutase 2, mitochondrial; TAC, transverse aortic constriction; TMX, thioredoxin-related transmembrane protein; XDH, xanthine dehydrogenase.
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    mitochondrially-targeted antioxidant mitoq  (MitoQ Ltd)
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    MitoQ Ltd mitochondrially-targeted antioxidant mitoq
    Transcriptional reprogramming of ROS signaling in c Sirt4 -Tg mice following TAC. Changes in expression of genes encoding for proteins involved in A) ROS production as determined by RNA-sequencing (n = 5–6) and B) validation of Nox4 expression by RT-qPCR ( n = 9–10); and C) <t>antioxidant</t> stress defense as determined by RNA-sequencing (n = 5–6) and D) validation of Gpx1 expression by RT-qPCR ( n = 9–10) in hearts of Control and c Sirt4 -Tg mice 12 weeks following TAC surgery with or without MitoQ supplementation. 2-Way ANOVA: #, effect of genotype; §, effect of treatment; %, effect of interaction. * p < 0.05; **p < 0.01; ***p < 0.001; ****p < 0.0001 using Fisher's LSD test. CAT, catalase; c Sirt4 -Tg, cardiomyocyte-specific overexpression of Sirt4 ; CYBA, cytochrome b -245, alpha chain; CYBB, cytochrome b -245, beta chain; GPX, glutathione peroxidase; MAOA, monoamine oxidase A; MAOB, monoamine oxidase B; MitoQ, mitoquinone; NOX4, NADPH oxidase 4; PRDX3, peroxiredoxin 3; SOD2, superoxide dismutase 2, mitochondrial; TAC, transverse aortic constriction; TMX, thioredoxin-related transmembrane protein; XDH, xanthine dehydrogenase.
    Mitochondrially Targeted Antioxidant Mitoq, supplied by MitoQ Ltd, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/mitochondrially-targeted antioxidant mitoq/product/MitoQ Ltd
    Average 90 stars, based on 1 article reviews
    mitochondrially-targeted antioxidant mitoq - by Bioz Stars, 2026-02
    90/100 stars
    		  Buy from Supplier

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    Transcriptional reprogramming of ROS signaling in c Sirt4 -Tg mice following TAC. Changes in expression of genes encoding for proteins involved in A) ROS production as determined by RNA-sequencing (n = 5–6) and B) validation of Nox4 expression by RT-qPCR ( n = 9–10); and C) antioxidant stress defense as determined by RNA-sequencing (n = 5–6) and D) validation of Gpx1 expression by RT-qPCR ( n = 9–10) in hearts of Control and c Sirt4 -Tg mice 12 weeks following TAC surgery with or without MitoQ supplementation. 2-Way ANOVA: #, effect of genotype; §, effect of treatment; %, effect of interaction. * p < 0.05; **p < 0.01; ***p < 0.001; ****p < 0.0001 using Fisher's LSD test. CAT, catalase; c Sirt4 -Tg, cardiomyocyte-specific overexpression of Sirt4 ; CYBA, cytochrome b -245, alpha chain; CYBB, cytochrome b -245, beta chain; GPX, glutathione peroxidase; MAOA, monoamine oxidase A; MAOB, monoamine oxidase B; MitoQ, mitoquinone; NOX4, NADPH oxidase 4; PRDX3, peroxiredoxin 3; SOD2, superoxide dismutase 2, mitochondrial; TAC, transverse aortic constriction; TMX, thioredoxin-related transmembrane protein; XDH, xanthine dehydrogenase.

    Journal: Journal of Molecular and Cellular Cardiology Plus

    Article Title: Sirtuin 4 accelerates heart failure development by enhancing reactive oxygen species-mediated profibrotic transcriptional signaling

    doi: 10.1016/j.jmccpl.2025.100299

    Figure Lengend Snippet: Transcriptional reprogramming of ROS signaling in c Sirt4 -Tg mice following TAC. Changes in expression of genes encoding for proteins involved in A) ROS production as determined by RNA-sequencing (n = 5–6) and B) validation of Nox4 expression by RT-qPCR ( n = 9–10); and C) antioxidant stress defense as determined by RNA-sequencing (n = 5–6) and D) validation of Gpx1 expression by RT-qPCR ( n = 9–10) in hearts of Control and c Sirt4 -Tg mice 12 weeks following TAC surgery with or without MitoQ supplementation. 2-Way ANOVA: #, effect of genotype; §, effect of treatment; %, effect of interaction. * p < 0.05; **p < 0.01; ***p < 0.001; ****p < 0.0001 using Fisher's LSD test. CAT, catalase; c Sirt4 -Tg, cardiomyocyte-specific overexpression of Sirt4 ; CYBA, cytochrome b -245, alpha chain; CYBB, cytochrome b -245, beta chain; GPX, glutathione peroxidase; MAOA, monoamine oxidase A; MAOB, monoamine oxidase B; MitoQ, mitoquinone; NOX4, NADPH oxidase 4; PRDX3, peroxiredoxin 3; SOD2, superoxide dismutase 2, mitochondrial; TAC, transverse aortic constriction; TMX, thioredoxin-related transmembrane protein; XDH, xanthine dehydrogenase.

    Article Snippet: At six weeks after TAC, control and c Sirt4 -Tg mice were treated with the mitochondrially targeted antioxidant MitoQ, provided by the company MitoQ (Auckland, NZ).

    Techniques: Expressing, RNA Sequencing, Biomarker Discovery, Quantitative RT-PCR, Control, Over Expression